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Biomax Inc human colorectal cancer tissue microarray
The level of Bmi1 expression dictates the metastatic potential of <t>colorectal</t> cancer (CRC) cells. (A) Formalin-fixed paraffin-embedded samples from four pairs of CRC patients were stained with the anti-Bmi1 antibody (magnification, 200×). (B) Bmi1 expression was evaluated via western blotting using anti-Bmi1 antibodies in SW480 and SW620 cells. (C) Migration of SW620 cells and Bmi1-depleted cells in zebrafish. (D) Expression of Bmi1 was analyzed via western blotting using anti-Bmi1 antibodies in suspended SW620 cells at specified time points.
Human Colorectal Cancer Tissue Microarray, supplied by Biomax Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/human colorectal cancer tissue microarray/product/Biomax Inc
Average 90 stars, based on 1 article reviews
human colorectal cancer tissue microarray - by Bioz Stars, 2026-04
90/100 stars

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1) Product Images from "Targeting Bmi1 for Enhancing Anoikis Sensitivity and Inhibiting Metastasis in Colorectal Cancer"

Article Title: Targeting Bmi1 for Enhancing Anoikis Sensitivity and Inhibiting Metastasis in Colorectal Cancer

Journal: Cancer Genomics & Proteomics

doi: 10.21873/cgp.20469

The level of Bmi1 expression dictates the metastatic potential of colorectal cancer (CRC) cells. (A) Formalin-fixed paraffin-embedded samples from four pairs of CRC patients were stained with the anti-Bmi1 antibody (magnification, 200×). (B) Bmi1 expression was evaluated via western blotting using anti-Bmi1 antibodies in SW480 and SW620 cells. (C) Migration of SW620 cells and Bmi1-depleted cells in zebrafish. (D) Expression of Bmi1 was analyzed via western blotting using anti-Bmi1 antibodies in suspended SW620 cells at specified time points.
Figure Legend Snippet: The level of Bmi1 expression dictates the metastatic potential of colorectal cancer (CRC) cells. (A) Formalin-fixed paraffin-embedded samples from four pairs of CRC patients were stained with the anti-Bmi1 antibody (magnification, 200×). (B) Bmi1 expression was evaluated via western blotting using anti-Bmi1 antibodies in SW480 and SW620 cells. (C) Migration of SW620 cells and Bmi1-depleted cells in zebrafish. (D) Expression of Bmi1 was analyzed via western blotting using anti-Bmi1 antibodies in suspended SW620 cells at specified time points.

Techniques Used: Expressing, Formalin-fixed Paraffin-Embedded, Staining, Western Blot, Migration

Bmi1 regulates anoikis resistance in colorectal cancer (CRC) cells. (A) Migration of SW620 cells and Bmi1-depleted cells was assessed using a Transwell assay after 24 h of growth in suspension. (B) The percentage of apoptotic SW620 cells and Bmi1-depleted cells at specified time points in suspension was determined by dual staining with annexin V-FITC and PI. The experiment was replicated thrice, and between-group comparisons were performed using a one-way ANOVA (*p<0.05). (C) The percentage of apoptotic SW620 cells treated with the Bmi1 inhibitor PTC209 at specified time points in suspension was determined by dual staining with annexin V-FITC and PI. The experiments were replicated thrice, and between-group comparisons were performed using a one-way ANOVA (*p<0.05). (D) Expression levels of Bmi1 and cleaved caspase-3 were assessed via western blotting after 48 h of suspension.
Figure Legend Snippet: Bmi1 regulates anoikis resistance in colorectal cancer (CRC) cells. (A) Migration of SW620 cells and Bmi1-depleted cells was assessed using a Transwell assay after 24 h of growth in suspension. (B) The percentage of apoptotic SW620 cells and Bmi1-depleted cells at specified time points in suspension was determined by dual staining with annexin V-FITC and PI. The experiment was replicated thrice, and between-group comparisons were performed using a one-way ANOVA (*p<0.05). (C) The percentage of apoptotic SW620 cells treated with the Bmi1 inhibitor PTC209 at specified time points in suspension was determined by dual staining with annexin V-FITC and PI. The experiments were replicated thrice, and between-group comparisons were performed using a one-way ANOVA (*p<0.05). (D) Expression levels of Bmi1 and cleaved caspase-3 were assessed via western blotting after 48 h of suspension.

Techniques Used: Migration, Transwell Assay, Suspension, Staining, Expressing, Western Blot


Figure Legend Snippet: Correlation between Bmi1, MDK and clinicopathological parameters. in colorectal cancer.

Techniques Used:



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Biomax Inc human colorectal cancer tissue microarray
The level of Bmi1 expression dictates the metastatic potential of <t>colorectal</t> cancer (CRC) cells. (A) Formalin-fixed paraffin-embedded samples from four pairs of CRC patients were stained with the anti-Bmi1 antibody (magnification, 200×). (B) Bmi1 expression was evaluated via western blotting using anti-Bmi1 antibodies in SW480 and SW620 cells. (C) Migration of SW620 cells and Bmi1-depleted cells in zebrafish. (D) Expression of Bmi1 was analyzed via western blotting using anti-Bmi1 antibodies in suspended SW620 cells at specified time points.
Human Colorectal Cancer Tissue Microarray, supplied by Biomax Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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human colorectal cancer tissue microarray - by Bioz Stars, 2026-04
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U.S Biomax Inc human colorectal cancer tissue microarray slides
( A ) Two human <t>colorectal</t> cancer tissue <t>microarray</t> slides consist of 172 tissue samples were stained with anti-TMIGD1 antibody. Staining was scored as 0 (negative, <5% cells positive), 1+ (6-25% cells positive), 2+ (26-50% cells positive), and 3+ (>50% cells positive). The tumor differentiation was graded morphologically (grade 1, 2 and 3 as well, moderately and poorly differentiated). ( B ) The average staining intensities were compared using ANOVA with Tukey post-hoc test per tumor grade. ( C ) The average staining intensities were compared using ANOVA with Tukey post-hoc test per tumor differentiation. ( D ) Western blot analysis of TMIGD1 expression in human renal and colorectal cancer cell lines. ( E ) qPCR analysis of TMIGD1 expression in CRC cell lines (HT29, HCT116 and RKO). Human kidney epithelial cells, HK2 was used as a positive control.
Human Colorectal Cancer Tissue Microarray Slides, supplied by U.S Biomax Inc, used in various techniques. Bioz Stars score: 88/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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CapG expresses in the human <t>colorectal</t> carcinoma and normal specimens in tissue <t>microarray.</t> A tissue microarray was used to examine the expression of CapG by immunohistochemistry; the expression index of CapG in the human colon tissues was quantified by a pathologist, and scores of the specimens were also organized depending on the pathologic diagnosis with normal, colorectal carcinoma, and metastatic colorectal carcinoma. ∗ p < 0.05.
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SuperBioChips human colorectal cancer tissue microarrays
CapG expresses in the human <t>colorectal</t> carcinoma and normal specimens in tissue <t>microarray.</t> A tissue microarray was used to examine the expression of CapG by immunohistochemistry; the expression index of CapG in the human colon tissues was quantified by a pathologist, and scores of the specimens were also organized depending on the pathologic diagnosis with normal, colorectal carcinoma, and metastatic colorectal carcinoma. ∗ p < 0.05.
Human Colorectal Cancer Tissue Microarrays, supplied by SuperBioChips, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/human colorectal cancer tissue microarrays/product/SuperBioChips
Average 90 stars, based on 1 article reviews
human colorectal cancer tissue microarrays - by Bioz Stars, 2026-04
90/100 stars
  Buy from Supplier

Image Search Results


The level of Bmi1 expression dictates the metastatic potential of colorectal cancer (CRC) cells. (A) Formalin-fixed paraffin-embedded samples from four pairs of CRC patients were stained with the anti-Bmi1 antibody (magnification, 200×). (B) Bmi1 expression was evaluated via western blotting using anti-Bmi1 antibodies in SW480 and SW620 cells. (C) Migration of SW620 cells and Bmi1-depleted cells in zebrafish. (D) Expression of Bmi1 was analyzed via western blotting using anti-Bmi1 antibodies in suspended SW620 cells at specified time points.

Journal: Cancer Genomics & Proteomics

Article Title: Targeting Bmi1 for Enhancing Anoikis Sensitivity and Inhibiting Metastasis in Colorectal Cancer

doi: 10.21873/cgp.20469

Figure Lengend Snippet: The level of Bmi1 expression dictates the metastatic potential of colorectal cancer (CRC) cells. (A) Formalin-fixed paraffin-embedded samples from four pairs of CRC patients were stained with the anti-Bmi1 antibody (magnification, 200×). (B) Bmi1 expression was evaluated via western blotting using anti-Bmi1 antibodies in SW480 and SW620 cells. (C) Migration of SW620 cells and Bmi1-depleted cells in zebrafish. (D) Expression of Bmi1 was analyzed via western blotting using anti-Bmi1 antibodies in suspended SW620 cells at specified time points.

Article Snippet: A human colorectal cancer tissue microarray (CDA3, Biomax Inc. and Super Bio Chips Laboratories, Seoul, Republic of Korea) was utilized.

Techniques: Expressing, Formalin-fixed Paraffin-Embedded, Staining, Western Blot, Migration

Bmi1 regulates anoikis resistance in colorectal cancer (CRC) cells. (A) Migration of SW620 cells and Bmi1-depleted cells was assessed using a Transwell assay after 24 h of growth in suspension. (B) The percentage of apoptotic SW620 cells and Bmi1-depleted cells at specified time points in suspension was determined by dual staining with annexin V-FITC and PI. The experiment was replicated thrice, and between-group comparisons were performed using a one-way ANOVA (*p<0.05). (C) The percentage of apoptotic SW620 cells treated with the Bmi1 inhibitor PTC209 at specified time points in suspension was determined by dual staining with annexin V-FITC and PI. The experiments were replicated thrice, and between-group comparisons were performed using a one-way ANOVA (*p<0.05). (D) Expression levels of Bmi1 and cleaved caspase-3 were assessed via western blotting after 48 h of suspension.

Journal: Cancer Genomics & Proteomics

Article Title: Targeting Bmi1 for Enhancing Anoikis Sensitivity and Inhibiting Metastasis in Colorectal Cancer

doi: 10.21873/cgp.20469

Figure Lengend Snippet: Bmi1 regulates anoikis resistance in colorectal cancer (CRC) cells. (A) Migration of SW620 cells and Bmi1-depleted cells was assessed using a Transwell assay after 24 h of growth in suspension. (B) The percentage of apoptotic SW620 cells and Bmi1-depleted cells at specified time points in suspension was determined by dual staining with annexin V-FITC and PI. The experiment was replicated thrice, and between-group comparisons were performed using a one-way ANOVA (*p<0.05). (C) The percentage of apoptotic SW620 cells treated with the Bmi1 inhibitor PTC209 at specified time points in suspension was determined by dual staining with annexin V-FITC and PI. The experiments were replicated thrice, and between-group comparisons were performed using a one-way ANOVA (*p<0.05). (D) Expression levels of Bmi1 and cleaved caspase-3 were assessed via western blotting after 48 h of suspension.

Article Snippet: A human colorectal cancer tissue microarray (CDA3, Biomax Inc. and Super Bio Chips Laboratories, Seoul, Republic of Korea) was utilized.

Techniques: Migration, Transwell Assay, Suspension, Staining, Expressing, Western Blot

Journal: Cancer Genomics & Proteomics

Article Title: Targeting Bmi1 for Enhancing Anoikis Sensitivity and Inhibiting Metastasis in Colorectal Cancer

doi: 10.21873/cgp.20469

Figure Lengend Snippet: Correlation between Bmi1, MDK and clinicopathological parameters. in colorectal cancer.

Article Snippet: A human colorectal cancer tissue microarray (CDA3, Biomax Inc. and Super Bio Chips Laboratories, Seoul, Republic of Korea) was utilized.

Techniques:

( A ) Two human colorectal cancer tissue microarray slides consist of 172 tissue samples were stained with anti-TMIGD1 antibody. Staining was scored as 0 (negative, <5% cells positive), 1+ (6-25% cells positive), 2+ (26-50% cells positive), and 3+ (>50% cells positive). The tumor differentiation was graded morphologically (grade 1, 2 and 3 as well, moderately and poorly differentiated). ( B ) The average staining intensities were compared using ANOVA with Tukey post-hoc test per tumor grade. ( C ) The average staining intensities were compared using ANOVA with Tukey post-hoc test per tumor differentiation. ( D ) Western blot analysis of TMIGD1 expression in human renal and colorectal cancer cell lines. ( E ) qPCR analysis of TMIGD1 expression in CRC cell lines (HT29, HCT116 and RKO). Human kidney epithelial cells, HK2 was used as a positive control.

Journal: bioRxiv

Article Title: TMIGD1, a putative tumor suppressor, induces G2-M cell cycle checkpoint arrest in colon cancer cells

doi: 10.1101/2020.06.06.138057

Figure Lengend Snippet: ( A ) Two human colorectal cancer tissue microarray slides consist of 172 tissue samples were stained with anti-TMIGD1 antibody. Staining was scored as 0 (negative, <5% cells positive), 1+ (6-25% cells positive), 2+ (26-50% cells positive), and 3+ (>50% cells positive). The tumor differentiation was graded morphologically (grade 1, 2 and 3 as well, moderately and poorly differentiated). ( B ) The average staining intensities were compared using ANOVA with Tukey post-hoc test per tumor grade. ( C ) The average staining intensities were compared using ANOVA with Tukey post-hoc test per tumor differentiation. ( D ) Western blot analysis of TMIGD1 expression in human renal and colorectal cancer cell lines. ( E ) qPCR analysis of TMIGD1 expression in CRC cell lines (HT29, HCT116 and RKO). Human kidney epithelial cells, HK2 was used as a positive control.

Article Snippet: Two human colorectal cancer tissue microarray slides (US Biomax, catalog numbers BC05012a and BC05118a) consist of 172 tissue samples (72 on BC05012a and 100 on BC05118a) were stained with anti-TMIGD1 antibody.

Techniques: Microarray, Staining, Western Blot, Expressing, Positive Control

CapG expresses in the human colorectal carcinoma and normal specimens in tissue microarray. A tissue microarray was used to examine the expression of CapG by immunohistochemistry; the expression index of CapG in the human colon tissues was quantified by a pathologist, and scores of the specimens were also organized depending on the pathologic diagnosis with normal, colorectal carcinoma, and metastatic colorectal carcinoma. ∗ p < 0.05.

Journal: Analytical Cellular Pathology (Amsterdam)

Article Title: Capping Actin Protein Overexpression in Human Colorectal Carcinoma and Its Contributed Tumor Migration

doi: 10.1155/2018/8623937

Figure Lengend Snippet: CapG expresses in the human colorectal carcinoma and normal specimens in tissue microarray. A tissue microarray was used to examine the expression of CapG by immunohistochemistry; the expression index of CapG in the human colon tissues was quantified by a pathologist, and scores of the specimens were also organized depending on the pathologic diagnosis with normal, colorectal carcinoma, and metastatic colorectal carcinoma. ∗ p < 0.05.

Article Snippet: A human colorectal cancer-metastasis-normal tissue microarray slide (CDA3) was purchased from SuperBioChips Laboratories (Seoul, Korea).

Techniques: Microarray, Expressing, Immunohistochemistry, Biomarker Discovery

The mRNA and protein expression levels of CapG in four human colorectal carcinoma cell lines. Four human CRC cell lines including HT29, DLD-1, HCT116, and SW1116 were used to analyze the expressions of (a) mRNA and (b) protein of CapG. GAPDH was used as a loading control.

Journal: Analytical Cellular Pathology (Amsterdam)

Article Title: Capping Actin Protein Overexpression in Human Colorectal Carcinoma and Its Contributed Tumor Migration

doi: 10.1155/2018/8623937

Figure Lengend Snippet: The mRNA and protein expression levels of CapG in four human colorectal carcinoma cell lines. Four human CRC cell lines including HT29, DLD-1, HCT116, and SW1116 were used to analyze the expressions of (a) mRNA and (b) protein of CapG. GAPDH was used as a loading control.

Article Snippet: A human colorectal cancer-metastasis-normal tissue microarray slide (CDA3) was purchased from SuperBioChips Laboratories (Seoul, Korea).

Techniques: Expressing, Control